Home / Dry Eye Assessment
Assessment · Your starting point
The Comprehensive Dry Eye Assessment
Dry eye isn’t one disease — it’s a spectrum of conditions with different causes and different treatments. The only way to treat it effectively is to diagnose what’s actually happening on your ocular surface. That’s what this appointment is for. Every UDEI patient begins here, no matter where they’ve been before.
your first assessment
measurements per visit
we differentiate
to your findings
Why it matters
Dry Eye Isn’t One Disease
A comprehensive dry eye assessment is the foundation of every effective treatment plan — because dry eye isn’t one disease. It’s a spectrum of conditions with different underlying causes, and a treatment that is right for one patient can be useless, or counterproductive, for another.
- Aqueous-deficient dry eye — your tears don’t have enough water to keep the surface lubricated
- Evaporative dry eye — your tears evaporate too quickly, usually because of dysfunctional meibomian glands
- Mixed dry eye — a combination of aqueous deficiency and evaporative components
- Ocular surface disease — inflammation or structural damage to the cornea, conjunctiva, or lid margin
- Secondary dry eye — related to systemic conditions like Sjögren’s syndrome, rheumatoid arthritis, or graft-versus-host disease
Treatment depends on diagnosis. A patient with aqueous-deficient dry eye needs a different plan than one with meibomian gland dysfunction. If the underlying picture is wrong or incomplete, the treatment will be too — which is why so many patients cycle through drop after drop with only partial improvement. That’s what the assessment exists to change.
A typical dry eye patient arrives at UDEI having tried several over-the-counter drops, cycled through prescription products, and spent months or years unsure why none of it really worked. The issue is almost never that the treatments were bad — it’s that they were chosen without a complete picture of what was actually happening on the ocular surface. The assessment changes the starting point.
Why this matters at UDEI
We don’t skip this step. Every new UDEI patient receives a full diagnostic workup regardless of how thorough their previous care has been. We’d rather take the extra hour now than spend months recommending treatments that don’t match the underlying pathology. Our team has done this assessment thousands of times, and that experience is what allows us to read subtle findings most clinics miss — and to spare patients from treatments they don’t actually need.
What’s included
What Happens During Your Assessment
Your first visit runs between 45 minutes and an hour. It combines a structured history with a sequence of objective diagnostic measurements, each of which tells us something specific about how your ocular surface is behaving. None of it is guesswork.
Patient history & symptom review
The appointment starts with a structured conversation. When did your symptoms begin? What makes them better or worse? Have you had eye surgery — cataract, LASIK, PRK, or SMILE? What drops and medications have you tried, and for how long? Any systemic conditions, autoimmune disease, or hormonal changes? Screen time, workplace environment, contact lens wear. Each answer shifts which diagnoses become more or less likely and shapes every test that follows.
Tear osmolarity testing
Osmolarity measures the salt concentration of your tears. A healthy tear film sits between 275 and 307 mOsm/L. Higher readings — or a significant difference between the two eyes — tell us the surface is stressed, even when symptoms are mild or ambiguous. Osmolarity is one of the most objective dry eye measurements available and guides whether we focus on conserving tears, improving their quality, or both.
Tear break-up time (TBUT)
We place a small amount of fluorescein dye in each eye and time how long the tear film stays stable after a blink. Normal is 15 seconds or more. Under 5 seconds means the tear film is unstable — a classic sign of evaporative dry eye, typically tied to meibomian gland dysfunction or an unhealthy lipid layer.
Ocular surface staining
Fluorescein and lissamine green dyes highlight damaged cells on the cornea and conjunctiva. The pattern, severity, and distribution of staining tell us where the surface has broken down and how advanced the disease is. Staining patterns are also how we track your response to treatment on follow-up visits — so progress (or lack of it) is visible, not just a feeling.
Meibomian gland evaluation (meibography)
We use infrared imaging to photograph the oil-producing glands in your eyelids — their shape, their volume, and whether any have atrophied or dropped out. We then express the glands manually to assess the quality of oil they release: clear, cloudy, thick, or absent. Gland dysfunction is by far the most common cause of evaporative dry eye, and these two measurements tell us exactly how far it has progressed.
Lipid layer analysis
Your tears have three layers — water, oil, and mucus — and the outermost oil layer is what prevents rapid evaporation. A thin or poor-quality lipid layer can leave you dry even when tear production is normal. We measure the thickness and distribution of your lipid layer to understand whether meibomian restoration should be part of your plan, and how aggressive that restoration needs to be.
Anterior segment imaging
High-magnification photography documents the front of your eye in fine detail: lid margin health, conjunctival tissue, corneal clarity, the size of your tear meniscus, and any secondary findings like pinguecula, pterygium, or blepharitis. This imaging becomes the baseline we compare against on every follow-up, so we can see exactly whether you’re improving, plateauing, or progressing.
Corneal topography
A topographic map of your cornea documents its shape, regularity, and any structural irregularities — scarring, ectasia, prior surgical changes. This is less about diagnosing dry eye itself and more about detecting the conditions that can mimic or worsen dry eye symptoms, and about establishing a baseline in case surgical or optical interventions are ever considered.
Not every patient needs every test at every visit — the workup is calibrated to your presentation, and some measurements are only repeated as follow-up when the clinical question requires it. But every new UDEI patient receives the core diagnostics, because that is what makes a treatment plan worth the paper it’s written on.
Primary mechanisms
What’s Actually Causing It
A good assessment doesn’t just confirm that you have dry eye — it identifies which underlying mechanism is driving your symptoms. These are the five primary causes we diagnose at UDEI, each with a different treatment pathway and prognosis. Most patients have more than one of them at the same time.
Meibomian gland dysfunction (MGD)
The most common cause of evaporative dry eye. Oil-producing glands along the lid margin become blocked, atrophied, or inflamed, and the tear film evaporates too quickly. Often present for years before diagnosis.
EvaporativeLearn about MGD →
Aqueous deficiency
The lacrimal glands don’t produce enough watery tear volume. May be age-related, autoimmune, or secondary to medication. The classic “my eyes burn and feel sandy” presentation — and the form of dry eye most responsive to tear conservation.
Aqueous-deficientLearn about aqueous deficiency →
Blepharitis & Demodex
Chronic inflammation of the lid margin — often driven by bacterial overgrowth or Demodex mites — that disrupts the tear film, worsens MGD, and contributes to persistent ocular surface disease. Treatable, but frequently missed.
Lid marginLearn about blepharitis →
Chalazion
A blocked and inflamed meibomian gland that forms a firm lump on the lid. Often a visible consequence of underlying gland dysfunction — and a reason to treat the whole lid margin, not just the single lump.
LidLearn about chalazion →
Allergic & toxic conjunctivitis
Chronic exposure to allergens, preservatives in eye drops, or irritants can mimic dry eye symptoms or sit alongside them. We identify these because the treatment is different — and because continuing the wrong drops can make things worse.
ReactiveLearn more →
Many patients have more than one of these diagnoses at the same time — MGD plus preservative-induced toxicity, for instance, or aqueous deficiency alongside chronic blepharitis. Part of the value of a careful assessment is separating the contributors so each can be addressed in the right order.
Is this you?
Is any of this you?
The assessment itself is the same universal workup no matter why you’re here — but how we interpret and prioritise what we find depends on your clinical story. The coordination pathway, the questions we ask, and the order we approach your care in all shift depending on what brought you through the door. If any of the scenarios below describe you, there’s extra context worth reading before your visit.
Contact lenses & computer use
Lens wearers and heavy screen users are the most common population we see. The underlying Meibomian gland dysfunction that makes lenses uncomfortable is often the same process that brings patients through UELC’s door years later asking about laser vision correction. Address it now and both paths get easier.
LifestyleContact lens & screen dry eye →
Cataract surgery & dry eye
Dry eye is both a risk factor for and a consequence of cataract surgery. Identifying and treating the underlying cause before surgery improves biometry accuracy and recovery — backed by UVG’s own 1,074-patient outcomes study.
Pre & post-opCataract & dry eye →
Refractive surgery & dry eye
Many patients consider LASIK, PRK, or SMILE because they’ve become contact-lens intolerant — and the same Meibomian gland dysfunction that made their lenses uncomfortable can follow them through laser if it isn’t treated first. Pre-optimising the surface and managing the 20–30% who experience persistent post-LASIK dryness. Coordinated with UELC.
Pre & post-opRefractive surgery & dry eye →
Glaucoma therapy & dry eye
Long-term use of preserved glaucoma drops is one of the most under-recognised causes of ocular surface disease. We work with your glaucoma specialist to protect both your nerve and your surface — often by adjusting formulations rather than starting over.
Drop-relatedGlaucoma & dry eye →
If more than one applies — a cataract patient who also wears contact lenses, say, or a glaucoma patient preparing for cataract surgery — pick whichever matches your most immediate concern. The assessment covers all of it regardless, and your report will reflect every context that applies to you.
What you leave with
Your Personalised Diagnostic Report
After your assessment, you’ll leave with a clear, written diagnostic report — a document you can keep, share with your optometrist, and refer back to as treatment progresses. No vague recommendations, no “try these drops and come back if it doesn’t work.”
- Your diagnosis — the root cause or causes of your dry eye, in plain language
- Severity classification — mild, moderate, or severe across each axis of disease
- Recommended treatment plan — which therapies are likely to help, in what order, at what cadence
- Expected timeline — how long improvement typically takes and what milestones to look for
- Home care protocol — drops, supplements, lid hygiene, and behavioural changes for daily use
- Follow-up schedule — when to return for treatment sessions and progress monitoring
The report goes home with you. If you have a referring optometrist, we’ll send them a copy automatically — so your care stays coordinated outside UDEI as well as inside. On follow-up visits, we compare your updated measurements against the baseline we established on day one, so you can see whether the plan is working — not just guess at it.
What comes next
Your Assessment Guides Your Treatment Plan
Your diagnosis tells us which treatments are likely to help — and which aren’t worth your time or money. Below are the core in-office and at-home interventions UDEI uses. Most patients receive two or three of these in combination, not just one.
UltraView DEL™ IPL therapy
Intense pulsed light treatment for meibomian gland dysfunction, ocular rosacea, and chronic inflammation. A course of in-office sessions restores gland function and reduces the inflammatory drivers of evaporative dry eye.
In-office procedureLearn about UltraView DEL →
Meibomian gland expression
Manual or device-assisted expression of clogged meibomian glands to restore a healthy lipid layer. Often combined with IPL or heat therapy, and typically repeated on a maintenance cadence.
In-office procedureLearn about gland expression →
Prescription dry eye drops
Anti-inflammatory drops — cyclosporine, lifitegrast, steroids in specific clinical pictures — that target the immune-driven component of chronic dry eye. Chosen based on your diagnosis, not defaulted to.
At-homeLearn about prescription drops →
Hemoderivatives (AS & ePRP)
Biologic eye drops made from your own blood, containing the growth factors your ocular surface uses to heal. Reserved for severe, refractory, autoimmune, or post-surgical dry eye — and produced in-house at UDEI.
Advanced therapyLearn about hemoderivatives →
Punctal plugs
Small, reversible plugs placed in the tear drainage ducts to conserve your own natural tears. A targeted option for patients with confirmed aqueous deficiency — not a default treatment for every dry eye patient.
In-office procedureLearn about punctal plugs →
Lid hygiene programme
Structured at-home care targeting blepharitis, Demodex, and lid margin inflammation. Often the unglamorous foundation of every successful dry eye plan — and the step patients most often skip without guided coaching.
At-homeLearn about lid hygiene →
The right combination depends on what the assessment finds — which is why we don’t prescribe any of it before we’ve looked. The goal is the minimum effective treatment plan that addresses your actual pathology, not the most expensive bundle we can sell.
Connected care
Dry Eye Is Rarely Just Dry Eye
Many patients arrive at UDEI with ocular surface problems that sit alongside other eye conditions — cataracts, post-laser recovery, glaucoma, or the need for specialty optical solutions. The UVG network lets us coordinate the full picture without sending you through a cold referral chain.
Developing cataracts alongside dry eye? Dry eye dramatically affects the accuracy of the measurements used to plan cataract surgery. Our Uptown Eye Specialists colleagues often send patients to UDEI first — so the surface is optimised before surgical planning, and visual outcomes reflect the eye the surgeon is actually working on.
Considering laser vision correction? A stable ocular surface is the single biggest predictor of laser vision correction comfort and outcomes. U Eye Laser Cosmetic routinely refers pre-operative patients to UDEI to confirm they’re good candidates before their refractive consultation — and post-operative patients with persistent dryness for advanced management.
Severe dry eye and can’t tolerate conventional contacts? Specialty scleral lenses vault over the cornea, trapping a fluid reservoir that keeps the ocular surface bathed in moisture all day. Patients whose dry eye makes soft contacts unwearable can often be fit with scleral lenses through our colleagues at U Optical.
Home-care products? Preservative-free artificial tears, omega-3 supplementation, lid-hygiene kits, and device-supported warm compresses that match what we recommend in-clinic are curated by our clinicians and available through U Shoppe.
Common questions
Frequently Asked Questions
Take the next step
The answer to “why are my eyes dry” starts here
If you’ve tried drops that didn’t work, cycled through prescriptions that never quite fit, or simply want to understand what’s actually going on, the comprehensive dry eye assessment is where to begin. Est. 2006, UDEI has served patients with severe and refractory ocular surface disease across the Greater Toronto Area.
